2 edition of Experimental renal hypertension found in the catalog.
Experimental renal hypertension
Page, Irvine H.
Bibliography: p. 63-64.
|Statement||by Irvine H. Page and Arthur Curtis Corcoran.|
|Series||American lecture series, no. 16. American lectures in physiology., American lecture series ;, no. 16.|
|LC Classifications||RC669 .P28 1948|
|The Physical Object|
|Pagination||v, 64 p. :|
|Number of Pages||64|
|LC Control Number||med48000049|
In response to hypertensive stimuli, mice lacking renal ACE do not produce renal angiotensin II. These studies indicate that the intrarenal renin–angiotensin system works as an entity separate from systemic angiotensin II generation. Renal ACE appears necessary for experimental hypertension. studies on experimental hypertension: ix. the effect on blood pressure of constriction of the abdominal aorta above and below the site of origin of both main renal arteries. j exp med. ; 69(5) (issn: ) goldblatt h; kahn jr; hanzal rf.
In patients with renal artery stenosis and renovascular hypertension, an abdominal bruit may be heard in the epigastrium, although the sound sometimes radiates to one side. 1 In one study of patients referred because of severe hypertension that was difficult to control—a setting suggesting renovascular hypertension—the finding of a systolic/diastolic abdominal bruit (i.e., continuous bruit. Book: All Authors / Contributors: Thomas F Lüscher; Norman M Kaplan. The Kidney and Regulation of Blood Pressure.- Glomerular Hemodynamics and Experimental Renal Injury.- Sodium, the Kidney, and Hypertension.- Epidemiology and Clinical Importance of Renovascular and Renal Parenchymatous Hypertension.- # Renal hypertension\/span>\n.
Renin in Experimental Renal Hypertension in Monkeys By Morton H. Frank, Ph.D. With the technical assistance of B. Keith and R. Speer • Although a role for renin in the pathogene-sis of acute and chronic experimental renal hypertension has been established in . Renovascular and renal excretory responses to intrarenally infused angiotensin II (Ang II) (1 and 3 ng/min, one dose per rat) were assessed in young (approximately 6 weeks of age) anesthetized spontaneously hypertensive rats (SHR) and Wistar-Kyoto (WKY) rats acutely treated with captopril. Urinary excretion of cyclic adenosine monophosphate (cAMP) was also measured in these rats to .
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Almost complete constriction of both main renal arteries, from the beginning, results in great elevation of systolic blood pressure which is accompanied by severe disturbance of renal function and uremia. This resembles the type of hypertension which is associated with so called malignant nephrosclerosis, in the sense of Fahr (17).Cited by: Additional Physical Format: Online version: Page, Irvine H.
(Irvine Heinly), Experimental renal hypertension. Springfield, Ill., C.C. Thomas . To regard the experimental type of hypertension as not exactly like human essential hypertension because the main renal artery of human beings with hyper- tension is not frequently stenotic, is to misunderstand the whole problem and the main purpose of the experimental procedures Experimental Renal Hypertension—Goldblatt which were used Cited by: Abstract.
This paper summarizes theoretical and experimental evidence which suggests that in Various experimental forms of hypertension, increased renal perfusion pressure is an essential circulatory compensation that allows normal excretion of salt and water despite reduced glomerular filtration or increased tubular reabsorption which tend to decrease renal by: 1.
the production of persistent hypertension in monkeys (macaque) by renal ischemia. j exp med. apr 30; 65 (5)– [pmc free article] goldblatt h, wartman wb.
studies on experimental hypertension: vi. the effect of section of anterior spinal nerve roots on experimental hypertension due to renal Cited by: Public Health Blood Pressure Hypertension Renal Function Renal Artery These keywords were added by machine and not by the authors.
This process is experimental and the keywords may be updated as the learning algorithm improves. The two-kidney, two-clip and one-kidney, one-clip models are volume-dependent models of hypertension [7, 8], with the former the animal equivalent of bilateral renal artery stenosis in humans.
It has been shown to be less dependent on the activation of the renin-angiotensin system (RAS) than the two-kidney, one-clip model. The Kidney: Morphology, Biochemistry, and Physiology, Volume IV covers the developments in the study of renal structure and function, particularly on the subcellular and molecular level.
This book is composed of six chapters that consider the correlation of kidney's structure with function, as well as the participation of the kidney in. experimental renal hypertension in rats (4) and dogs (5).
It has been suggested that increase in cardiac output might result from increase in venous return to the heart due to peripheral venous constriction (5, 6). Periph-eral venous constriction, by elevating capillary.
In this context, observations on experimental models of hypertension in which vessel wall elasticity is also reduced are of interest. 10 Using direct measurements of cell number and DNA measurements, Owens and Schwartz 11 12 examined the aorta of spontaneously hypertensive rats and rats with renovascular hypertension and found evidence for.
Purchase Pathophysiology of Kidney Disease and Hypertension - 1st Edition. Print Book & E-Book. ISBNOCLC Number: Description: viii, pages illustrations. Contents: Introduction: production of experimental renal hypertension by various methods --Production of experimental renal hypertension by constriction of main renal artery --Effect on blood pressure of moderate constriction of the main artery of only one kidney --Effect on blood pressure of moderate constriction of both main.
The relationship between hypertension, antihypertensive treatments and the risk of renal-cell cancer (RCC) remains still controversial.
To evaluate the strength of the evidence provided by the epidemiological literature on this topic, a MEDLINE search of. Experimental counterparts of these two clinical forms of renovascular hypertension can be found in the 1-kidney, 1-clip and the 2-kidney, 1-clip models of Goldblatt hypertension, respectively.
View chapter Purchase book. KOLFF WJ, PAGE IH. Persistence of experimental renal hypertension after total nephrectomy in dogs.
Am J Physiol. Sep; (3)– LEDINGHAM JM. The distribution of water, sodium, and potassium in heart and skeletal muscle in experimental renal hypertension in rats. Clin Sci. Nov; 12 (4)– LUXTON RW.
The same humoral mechanism which is responsible for experimental renal hypertension in the dog and other animals also obtains in the pathogenesis of experimental renal hypertension in the sheep and goat. Full Text.
The Full Text of this article is available as a PDF (K). High-intensity aerobic training lowers blood pressure and modulates the renal renin-angiotensin system in spontaneously hypertensive rats. Clinical and Experimental Hypertension: Vol. 42, No. 3. A significant amount of work within the traditional areas has been published, and several new dimensions are now being developed, mostly in the experimental setting.
These dimensions are discussed in several chapters of this new edition, The Kidney and Hypertension in Diabetes Mellitus, Fourth Edition. Introduction to the conference on experimental hypertension / William Goldring --Introductory lecture on the production and pathogenesis of experimental hypertension / Harry Goldblatt --Mechanism of renal hypertension / E.C.
Coke --Renin and renal hypertension / L.F. Leloir --The problem of the occurrence of vasoconstrictor substances in. Hypertension in end-stage renal disease. Patients with moderate to severe renal disease have a very high incidence of hypertension. In end-stage renal disease (ESRD) this is true regardless of the nature of the underlying renal disease.
Nevertheless, patients with glomerular diseases and autosomal dominant polycyctic kidney disease are particularly vulnerable. To investigate the hemodynamics of early experimental renal hypertension, skin and muscle blood flows and intravascular pressures were measured in the isolated, innervated, naturally perfused forelimbs of 44 male mongrel dogs under pentobarbital (35 mg/kg) anesthesia.
In addition, venous pressure-volume relationships were studied in temporarily.Treatment of experimental renal hypertension with renal extracts and extract fractions. WAKERLIN CE, KAMM O.
Proc Annu Meet Cent Soc Clin Res U S,01 .1. According to the renal body fluid feedback mechanism for long‐term control, persistent hypertension can only occur as a result of a reduction in renal sodium excretory function or a hypertensive shift in the pressure natriuresis relationship.